6 edition of The Physiologic, metabolic, and immunologic actions of interleukin-1 found in the catalog.
Includes bibliographies and index.
|Statement||editors, Matthew J. Kluger, Joost J. Oppenheim, Michael C. Powanda.|
|Series||Progress in leukocyte biology ;, v. 2|
|Contributions||Kluger, Matthew J., 1946-, Oppenheim, Joost J., 1934-, Powanda, M. C.|
|LC Classifications||QP801.I49 P49 1985|
|The Physical Object|
|Pagination||xxv, 559 p. :|
|Number of Pages||559|
|LC Control Number||85019688|
Single injections of recombinant human interleukin 1 beta (IL-1 beta) caused large (up to 2 degrees C) and sustained (3 h) increases in body temperature in conscious rats. Intracerebroventricular injections ( ng) were much more effective and elicited greater responses than intravenous injections ( . This book is currently out of print: sold out!. A second edition should be available in GI GIT Physiology BL Blood & Immunology EM Endocrine & Metabolic Physiology NU Neurophysiology MU Physiology of Muscle & Neuromuscular Junction MF Maternal, Foetal & Neonatal Physiology CM Clinical Measurement Coding LettersFile Size: KB.
the immune system is constituted by circulating cells in the blood and lymph, their collection in lymphoid organs, and cells scattered within most tissues. In physiological conditions, peripheral tissues contain mainly macrophages and dendritic cells, but other leukocytes (monocytes, neutrophils, lymphocytes) can transiently infiltrate tissues during pathological by: Physiology (/ ˌ f ɪ z i ˈ ɒ l ə dʒ i /; from Ancient Greek φύσις (physis), meaning 'nature, origin', and -λογία (-logia), meaning 'study of') is the scientific study of functions and mechanisms in a living system. As a sub-discipline of biology, physiology focuses on how organisms, organ systems, individual organs, cells, and biomolecules carry out the chemical and physical.
The Biology and Physiology of Inflammation As you learned in the “Immune System” module, there is an intimate relationship between the mechanism of inflammation and the immune system response. Inflammation is the body’s normal physiological response to Size: KB. Although far from conclusive, emerging evidence suggests that inflammation in the central and peripheral immune system may link metabolic syndrome to major depressive disorder. In this review, we will synthesize available data supporting this view and identify critical areas for future by:
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Physiologic, metabolic, and immunologic actions of interleukin New York: Liss, © (OCoLC) Material Type: Conference publication: Document Type: Book: All Authors / Contributors: Matthew J Kluger; Joost J Oppenheim; M C Powanda. Abstract. During infection, inflammation, or trauma, many phagocytic cells release interleukin-l, a protein responsible for initiating dozens of physiologic, metabolic and immunologic : M.
Kluger. Activation of the immune system, such as that produced by infection, may act as a physiological stressor causing activation of the adrenal cortex and increasing circulating glucocorticoid levels.
Stimulation of macrophages and monocytes causes a release of interleukin-1 (IL-1), which is a protein with a wide variety of effects including. Interleukin 1 (DL1) in rheumatoid arthritis: Synovial and blood dendritic cell production of IL In Kluger, MJ, Oppenheim, JJ and Powanda, MC (eds) The Physiologic, Metabolic and Immunologic Actions of Interleukin-1, pp.
– (New York: Alan R Liss) Google ScholarAuthor: Michael C. Powanda, Elizabeth D. Moyer. Cell sources and inducers of IL-1 are discussed, as are its chemical nature and mechanisms of action.
In addition, the importance of IL-1 and its effects on host defense mechanisms are presented. For example, how ILmediated responses, such as elevated temperature, lymphocyte activation, and systemic metabolic changes, alter the host as well.
Among these factors interleukin-1 has been particularly associated with both the systemic and local components of inflammation, since it shows a broad spectrum of biological activities involving all phases of the physiological response to disease and : Steven K.
Dower. The physiologic, metabolic and immunologic action of interleukin-1, New York: Alan Liss Inc, Brown K M, Rosenstreich D L. Mechanism of action of a human interleukin-1 by: 4. Interleukin-1 and its receptor antagonist in inflammatory arthritis.
The role of cytokines in RA, and the rationale for inhibition of IL-1 and tumor necrosis factor (TNF)-α in this disease have been extensively reviewed [6,7].Much evidence indicates that both of these proinflammatory cytokines are overproduced in the rheumatoid joint and are key mediators in both inflammation and tissue Cited by: Immunology (74) Insulin Action-Adipocyte Biology (21) Insulin Action-Cellular and Molecular Metabolism (15) Insulin Action-Glucose Transport and Insulin Resistance In Vitro (2) Insulin Action-Signal Transduction, Integrated Physiology-Macronutrient Metabolism and Food Intake (10) Integrated Physiology-Muscle (19).
W.P. Arend, F.G. Joslin, R.J. MassoniEffects of immune complexes on production of human monocytes of interleukin-1 or an interleukin-1 inhibitor J.
Immunol., (), pp. Google ScholarCited by: Brain Research, () Elsevier BRES Action site of circulating interleukin-1 on the rabbit brain M.
Hashimoto1, Y. Ishikawa2, S. Yokota3, F. Goto4, T. Bando5, Y. Sakakibara~ and M. Iriki1 1Department of Physiology, 21nstitule for Experimental Animals, 3Department of Anatomy, Yamanashi Medical College, Tamaho, Yamanashi, 4Department of Pathology, Kumamoto University Cited by: Dunn CJ, Fleming WE () The role of interleukin-1 in the inflammatory response with particular reference to endothelial cell-leukocyte adhesion.
In: Kluger MJ, Oppenheim JJ, Powanda MC (eds) The physiologic, metabolic, and immunologic actions of interleukin Liss, New York, pp Author: C. Dinarello. Title:Physiologic Versus Diabetogenic Effects of Interleukin A Question of Weight VOLUME: 20 ISSUE: 29 Author(s):Hugo O.
Besedovsky and Adriana del Rey Affiliation:Research Group Immunophysiology, Division of Neurophysiology, Inst. of Physiology and Pathophysiology, Deutschhausstrasse 2, Marburg, Germany.
Keywords:IL-1, physiologic functions, glucose Cited by: 9. J Leukoc Biol. Jun;37(6) Program and abstracts for a symposium on the physiologic, metabolic, and immunologic actions of interleukin Kunkel SL, Chensue SW, Spengler M, Geer JEffects of arachidonic acid metabolites and their metabolic inhibitors on interleukin-1 production Kluger MJ, Oppenheim JJ, Powanada MC (Eds.), The Physiologic, Metabolic, and Immunologic Actions of Interleukin-1, Alan R.
Liss, New York (), pp. Cited by: 2. Journal of Leukocyte Biology considers manuscripts of original investigations focusing on the origins, developmental biology, biochemistry and functions of granulocytes, lymphocytes, mononuclear phagocytes, and other cells involved in host defense. These include full-length papers on original research, rapid communications of new discoveries, letters, commentaries, and invited reviews.
The Fourth Edition of Microbial Physiology retains the logical, easy-to-follow organization of the previous editions. An introduction to cell structure and synthesis of cell components is provided, followed by detailed discussions of genetics, metabolism, growth, and regulation for anyone wishing to understand the mechanisms underlying cell survival and growth.
Filkins JP, Mathews HL, Yelich MR () Monokines and hyperinsulinism in endotoxicosis and septic peritonitis. In: Kluger MJ, Oppenheim JJ, Powanda MC (eds) The physiologic, metabolic, and immunologic actions of interleukin Liss, New York, pp – Google ScholarCited by: 5.
Genes and Development, ISSN11/, Vol Is pp. - The cholinergic antiinflammatory pathway. Recently, my colleagues and I described an alternative mechanism for cytokine control based on the structure of the nervous system (Figure (Figure2) 2) ().We reasoned that, since the CNS coordinates major physiological responses via innervated circuits, it might also use neural input to control a potentially deadly cytokine response, especially one.
In addition to controlling cellular lipid metabolism and homeostasis, sterol regulatory element-binding proteins (SREBPs) act as nodes of convergence and divergence within myriad physiological and Cited by: In obesity and metabolic syndrome as well as in patients with overt diabetes the metabolic actions of IGF-I are altered significantly.
Similarly its ability to coordinate its functions with those of GH and insulin is impaired in this pathophysiologic state and this constitutes an important component of the maladaptive response of all 3 hormones Cited by: The interleukin‐1 (IL‐1) family of ligands and receptors plays a central role in the generation and regulation of inflammation as a part of the immune response.